Jason Wolfe's ATLS® Trauma Moulage Page
(Thoughts on the Management of the Multiply Injured Patient)
Aim : |
To give people a framework for thinking about the management of the
traumatised patient and assist them to pass the ATLS trauma moulage.
This webpage was produced as a culmination of the teaching and experiences I
gained during a past ATLS course. |
ATLS®: |
The ATLS course itself is an excellent way of practicing the theoretical and
practical aspects of trauma management. I highly recommend anyone involved
in management of trauma patients to do it. |
Note : |
This is not meant as a short cut which negates the need to read the ATLS
course manual. You are reminded that it is extremely unlikely that you will
pass the course if you don't read the ATLS manual. |
PS: |
Note that the term 'ATLS' is a registered trade mark of the 'American College of Surgeons'. This web-page is not affiliated to, nor officially endorsed by them. |
1. | There is a need for rapid evaluation of the trauma patient. Time wasted costs lives. |
2. | The absence of a definitive diagnosis should never impede the application of essential treatment. |
3. | The first 'Golden Hour' is crucial to both the short and long term survival of the patient. It also is also critical in determining the morbidity that the patient will endure. |
4. | There is a need to establish management priorities: The things which will kill the patient first are always the things which should be checked and treated first. Things which will kill the patient later are managed later. Thus, airway problems are managed and treated before breathing problems, which in turn are treated before circulatory problems. |
5. | Patients with serious injuries are managed by the 'Trauma Team'. This will normally include ED doctors, an anaesthetist, along with specialists in the particular injury involved. Eg. Thoracic Surgeons / Orthopaedics / General Surgeons / Neurosurgeons. |
6. | All treatment modalities should be governed by the abiding principle of 'First do no harm'. |
(Stages & Subject Headings)
1. | Preparation |
2. | Triage |
3. | Primary Survey (ABCDE) & Resuscitation |
4. | Adjuncts to Primary Survey & Resuscitation |
5. | Consider need for Patient Transfer |
6. | Secondary Survey (with AMPLE History) |
7. | Continued Post-Resuscitation Monitoring & Re-evaluation |
8. | Transfer to Definitive Care |
1. | Pulse Oximeter |
2. | Blood Pressure |
3. | Cardiac Monitor / Electrocardiogram |
4. | Arterial Blood Gases / End Tidal pCO2 |
5. | X-Rays - Chest X-Ray / Cervical Spine / Pelvis / Others |
6. | FAST Ultrasound (Focused Assessment with Sonography for Trauma) |
7. | Nasogastric Tube & Urinary Catheter |
8. | Core Temperature |
1. | Inadequate Airway Protection |
2. | Airway Obstruction |
3. | Tension Pneumothorax |
4. | Open pneumothorax |
5. | Flail Chest with Hypoxia |
6. | Massive Haemothorax |
7. | Cardiac Tamponade |
8. | Severe Hypothermia |
9. | Severe Shock from Haemorrhage Unresponsive to Fluid Resuscitation. |
1. | Simple Pneumothorax |
2. | Haemothorax |
3. | Pulmonary Contusion |
4. | Tracheo-Bronchial Injury |
5. | Blunt Cardiac Injury |
6. | Traumatic Aortic Disruption |
7. | Diaphragmatic Rupture |
8. | Mediastinal Traversing Wounds |
9. | Blunt Oesophageal Trauma |
10. | Sternal / Scapular / Rib Fractures |
11. | Ruptured Liver or Spleen |
12. | Rupture of an abdominal or pelvic viscus |
13. | Any other chest / abdominal / or pelvic injuries which have resulted in organ damage but not in immediate shock |
1. | Go back and check Airway & Breathing. |
2. | The patient could be BLEEDING faster than you are replacing blood. These patients need to be taken to theatre immediately for surgical repair of the injured organ or vessel. Patients with severe blood loss & shock may be considered suitable for early administration of Tranexamic Acid. Consider activating the 'Massive Transfusion Protocol'. |
3. | The patient could be HYPOTHERMIC and therefore may be responding more slowly than a normothermic patient. |
4. | The patient could be in CARDIOGENIC SHOCK: Here the heart pump is failing due to blunt trauma, or sometimes due to penetrating trauma. Consider again pericardial tamponade and act appropriately if required. Consider early CVP monitoring. |
5. | The patient may be PREGNANT. If moderately or heavily pregnant women are treated in the supine position, the bulky uterus may impede the flow of blood in the Inferior Vena Cava. Such patients should be bolstered so that they are lying slightly on their left side by placing sand-bags or pillows under the right side of the pelvis and chest. This manoeuvre should be carried out earlier rather than later in the resuscitation. |
6. | The patient may be in NEUROGENIC SHOCK: This occurs with spinal cord injuries in which the sympathetic outflow is damaged. This denervation of the heart and blood vessels results in a clinical picture of hypotension without tachycardia or peripheral vasoconstriction. Volume resuscitation is still the primary treatment, but consideration should be given to the judicious use of vasopressors. Early CVP monitoring & Swan-Ganz pulmonary artery catheterisation may also be useful. |
7. | SEPTIC SHOCK: This is uncommon in the early period following trauma but may occur in penetrating abdominal injuries with a perforated viscus or in other penetrating injuries where the wound has been contaminated with dirty exogenous debris, especially if arrival in A&E has been delayed for hours or days. It is identified by the presence of hypotension, tachycardia, pyrexia and cutaneous vasodilation. |
1. | OLD AGE - Elderly patients have less 'physiological reserve': They are less able to increase heart rate and stroke volume in response to shock. Vital organs are more sensitive to the decreased blood flow and hypoxia associated with shock. The lungs are less efficient at the gaseous exchange of oxygen. The kidney is less able to respond to the volume preserving stimulus of the stress hormones Aldosterone, Anti-Diuretic Hormone & Cortisol. All these facts contribute to its increased morbidity and mortality. It is thus even more crucial in the elderly patient to pay meticulous attention to volume resuscitation, and the placement of arterial and CVP invasive monitoring devices will greatly assist in its assessment. These devices should be placed earlier rather than later. |
2. | YOUNG AGE - Children and babies have an especially high physiological reserve. Homeostatic mechanisms maintain blood pressure and cardiac output despite the loss of large percentages of their blood volume. However when the percentage of blood loss gets to about 40% (Class IV haemorrhage), the blood pressure and cardiac output drop precipitously. The lesson here is that children may still have normal vital observations despite being in a high level of shock. Always take advice from a paediatrician early. |
3. | ATHLETES - Althletes may have an increased blood volume of up to 15 - 20%, stroke volume can increase by 50%, cardiac output can increase by 600% and resting pulse is generally lower than unfit individuals. These facts mean that the usual clinical signs of hypovolaemia may not be manifested in athletes, even though significant blood loss may have occurred. |
4. | PREGNANCY - Women have a higher plasma volume during pregnancy. Cardiac output increases by 1.0 - 1.5 litres / minute, and heart rate increases by 10 - 15 beats / minute. Minute ventilation increases also (primarily due to an increase in the respiratory tidal volume), and the Renal Glomerular Filtration Rate also increases. All these things increase the physiological reserve of the mother and mean that signs of hypovolaemia appear later. The physiological responses to shock will always favour the mother, and whereas even in moderate shock, the mother may be quite well, the foetus may actually be in severe shock, deprived of the majority of its perfusion. Invasive maternal monitoring and foetal cardiotocographic monitoring are often required at an early stage to minimise complications to both mother and foetus. Always take advice from an obstetrician early. |
5. | DRUGS - Various drugs can affect the body's response to stress. Beta- blockers prevent the tachycardia and increased sympathetic responses to shock and may confuse the clinical picture. Diuretic use causes a relative hypovolaemia which may impair the body's reserve to respond to stress. |
6. |
HEAD INJURIES - The brain has a very high demand for oxygen and so secondary
brain damage will occur very quickly if the brain is deprived of its supply
of oxygenated blood. The Cerebral Perfusion Pressure is equal to the Mean
Arterial Blood Pressure minus the Intra-Cranial Pressure. Thus, brain
perfusion is reduced either by a decrease in blood pressure, or by an
increase in intra-cranial pressure. Head injuries may increase
intra-cranial pressure by the presence of mass-lesions (haematoma)
preventing the free circulation of cerebro-spinal fluid. Sub-arachnoid
haemorrhage increases intra-cranial pressure because the blood in the
cerebro-spinal fluid blocks the arachnoid granulations and thereby stops
the CSF from being reabsorbed back into the venous system. There are a number of conflicting processes in the head injured patient that make it essential to treat shock and hypovolaemia in a very precise manner. Over cautious volume resuscitation will result in hypotension wheras over enthusiastic volume resuscitation will result in volume overload which may exacerbate an already precarious intra-cranial pressure. The key aspects in the optimal management of the head injured patient include : early invasive monitoring to assist in accurate volume resuscitation, early endotracheal intubation to assist with hyperventilation (aim low end of normal pCO2), and early consultation with an experienced neurosurgeon. |
Here endeth the lesson !!
To contact me : Email
My Website is at http://www.jwolfe.clara.net.
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Last Modified : 12th January 2019